which of the following is a primary intervention for managing hyperphosphatemia
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Nursing Elites

ATI RN

ATI RN Custom Exams Set 4

1. Which of the following is a primary intervention for managing hyperphosphatemia?

Correct answer: D

Rationale: The correct answer is D, administering phosphate binders. Phosphate binders are a primary intervention for managing hyperphosphatemia as they help by binding phosphorus in the gut, preventing its absorption. Increasing calcium intake (choice A) or phosphorus intake (choice B) would exacerbate hyperphosphatemia. Decreasing calcium intake (choice C) is not a primary intervention for managing high phosphorus levels.

2. One of the reasons hospital patients are at greater risk for drug-nutrient interactions than they used to be is because:

Correct answer: A

Rationale: The correct answer is A. Hospitalized patients are more acutely ill, often having multiple conditions and treatments, which increases the risk of drug-nutrient interactions. Choice B is incorrect because hospital routines do not specifically interfere with the timing of medications in relation to drug-nutrient interactions. Choice C is incorrect because the toxicity and side effects of drugs do not directly relate to an increased risk of drug-nutrient interactions. Choice D is incorrect as sharing responsibility for monitoring does not inherently increase the risk of drug-nutrient interactions in hospital patients.

3. Which laboratory data indicate the client’s pancreatitis is improving?

Correct answer: A

Rationale: The correct answer is A. Amylase and lipase are enzymes specifically related to pancreatitis. A decrease in their serum levels indicates improvement in pancreatitis. White blood cell count (WBC), choices C and D, are not direct markers for pancreatitis improvement. Bilirubin levels, choice C, are more related to liver function rather than pancreatitis. Blood urea nitrogen (BUN) level, choice D, is a marker for kidney function, not pancreatitis.

4. Which nutrient deficiency is most likely to be seen in patients with chronic alcoholism?

Correct answer: D

Rationale: Patients with chronic alcoholism are most likely to develop a deficiency in Vitamin B1 (thiamine) due to poor dietary intake and impaired absorption. This deficiency can lead to conditions like Wernicke's encephalopathy and Korsakoff's syndrome. While deficiencies in other vitamins can also occur in chronic alcoholism, Vitamin B1 deficiency is more commonly associated with this condition, making it the most likely nutrient deficiency in these patients. Therefore, the correct answer is Vitamin B1 (Choice D). Deficiencies in Vitamin C (Choice A), Vitamin D (Choice B), and Vitamin B12 (Choice C) can also be seen in patients with chronic alcoholism, but they are not as commonly linked to this condition compared to Vitamin B1 deficiency.

5. Patients with gallbladder disease should reduce their intake of:

Correct answer: D

Rationale: Patients with gallbladder disease are advised to reduce their fat intake because fats can trigger gallbladder symptoms such as pain and bloating. While protein, sodium, and cholesterol may also impact overall health, reducing fat intake specifically helps manage gallbladder-related symptoms effectively. Protein is important for tissue repair, sodium can affect blood pressure, and cholesterol levels impact heart health, but in the context of gallbladder disease, fat reduction is the most beneficial.

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