Nursing Elites

1. During lactation, where is lactose synthesized in the body?

• A. Liver
• B. Breast
• C. Kidney
• D. Pancreas

Correct answer: B

Rationale: The correct answer is B: Breast. Lactose is synthesized in the breast during lactation to produce breastmilk. The liver is responsible for various metabolic functions but does not synthesize lactose. The kidney's primary role is in filtering blood and regulating electrolytes, not lactose synthesis. The pancreas is involved in producing digestive enzymes and insulin, not lactose synthesis.

2. Which hormone is responsible for stimulating the gallbladder to contract and release bile?

• A. gastric lipase
• B. cholecystokinin
• C. vitamin D
• D. enterokinin

Correct answer: B

Rationale: Cholecystokinin (CCK) is the correct answer. It is the hormone responsible for stimulating the gallbladder to contract and release bile to aid in fat digestion. Gastric lipase (Choice A) is an enzyme that breaks down fats in the stomach, not a hormone involved in gallbladder function. Vitamin D (Choice C) is important for calcium absorption and bone health, but it is not directly related to gallbladder function. Enterokinin (Choice D) is a peptide hormone that affects smooth muscle contractions in the gastrointestinal tract, but it is not specifically responsible for stimulating the gallbladder to release bile.

3. The type of protein-energy malnutrition that results in edema, hypoalbuminemia, skin lesions, and fatty liver is:

• A. cachexia.
• B. marasmus.
• C. kwashiorkor.
• D. sarcopenia.

Correct answer: C

Rationale: The correct answer is C, kwashiorkor. Kwashiorkor is a type of protein-energy malnutrition characterized by edema, hypoalbuminemia, skin lesions, and fatty liver. Edema is a key feature of kwashiorkor due to hypoalbuminemia, which leads to decreased oncotic pressure. Marasmus (choice B) is a form of severe malnutrition characterized by energy deficiency without significant protein deficiency, resulting in severe wasting. Cachexia (choice A) is a syndrome characterized by weight loss, muscle atrophy, fatigue, weakness, and loss of appetite in someone who is not actively trying to lose weight. Sarcopenia (choice D) is the age-related loss of muscle mass and function.

4. The lining of the stomach and intestine is protected from self-digestion by:

• A. pepsinogen.
• B. bile.
• C. mucus.
• D. fat.

Correct answer: C

Rationale: The correct answer is C: mucus. Mucus forms a protective barrier that prevents the stomach and intestine from digesting themselves. Pepsinogen (choice A) is an inactive form of pepsin, a digestive enzyme, and does not protect the stomach lining. Bile (choice B) aids in the digestion and absorption of fats but does not protect the stomach lining. Fat (choice D) is a nutrient and energy source; it does not play a role in protecting the stomach and intestine from self-digestion.

5. Which component of fat can be used to make glucose through gluconeogenesis?

• A. Glycogen
• B. Fatty acids
• C. Glycerol
• D. Monoglyceride

Correct answer: C

Rationale: The correct answer is 'Glycerol.' Glycerol, a component of fat, can be converted into glucose through gluconeogenesis. Glycogen (Choice A) is a polysaccharide stored in animals, not a component of fat. Fatty acids (Choice B) are used primarily for energy production through beta-oxidation, not for gluconeogenesis. Monoglyceride (Choice D) is a molecule formed during fat digestion, but it is not the component of fat that can be used to make glucose.

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