Nursing Elites

1. Ketones are produced when:

• A. carbohydrate stores are depleted.
• B. excess protein is consumed.
• C. protein stores are depleted.
• D. fat stores are depleted.

Correct answer: A

Rationale: Ketones are produced when the body uses fat for energy due to the depletion of carbohydrate stores. When carbohydrate stores are depleted, the body turns to fat as its primary source of energy, leading to the production of ketones. Excess protein consumption does not directly lead to ketone production. Similarly, the depletion of protein stores or fat stores is not directly related to the production of ketones.

2. The type of protein-energy malnutrition (PEM) that develops when the diet lacks protein is called:

• A. kwashiorkor.
• B. marasmus.
• C. anemia.
• D. beriberi.

Correct answer: A

Rationale: The correct answer is A: kwashiorkor. Kwashiorkor is a form of protein-energy malnutrition that arises due to a protein-deficient diet. This condition leads to symptoms such as edema, skin lesions, and fatty liver. Choice B, marasmus, is a form of PEM caused by overall malnutrition (protein and calorie deficiency), resulting in severe wasting and muscle loss. Choice C, anemia, is a condition characterized by a deficiency of red blood cells or hemoglobin, not specifically related to protein deficiency. Choice D, beriberi, is a disease caused by thiamine (Vitamin B1) deficiency, not a form of PEM related to protein deficiency.

3. Most fat replacers are made from:

• A. proteins.
• B. carbohydrates.
• C. cholesterol.
• D. monoglycerides.

Correct answer: B

Rationale: Most fat replacers are made from carbohydrates. Carbohydrates are utilized to mimic the texture of fats in food products. Proteins (Choice A) are not commonly used as fat replacers and are more often associated with other functions in food. Cholesterol (Choice C) is a type of fat and not used to replace fat in food products. Monoglycerides (Choice D) are sometimes used as emulsifiers or stabilizers in food products but are not the primary source of fat replacers.

4. How is resistant starch digested in the colon?

• A. bacterial fermentation.
• B. pancreatic amylase.
• C. hydrochloric acid.
• D. villi and microvilli.

Correct answer: A

Rationale: In the colon, resistant starch is digested by bacterial fermentation. The correct answer is A. During this process, short-chain fatty acids are produced. Pancreatic amylase, as mentioned in choice B, is responsible for breaking down starch in the small intestine, not in the colon. Choice C, hydrochloric acid, functions in the stomach to aid in the digestion of proteins, not starch. Villi and microvilli, as stated in choice D, are structures in the small intestine that absorb nutrients; they do not participate in the digestion of resistant starch in the colon.

5. What is the muscle layer on the outside of the intestinal wall called?

• A. serosa
• B. mucosa
• C. submucosa
• D. muscularis mucosae

Correct answer: A

Rationale: The correct answer is A: serosa. The serosa is the outermost layer of the intestinal wall, providing a protective covering. Choice B, mucosa, is the innermost layer responsible for absorption. Choice C, submucosa, lies beneath the mucosa and contains blood vessels and nerves. Choice D, muscularis mucosae, is a smooth muscle layer located within the mucosa, helping to create folds in the intestinal lining to increase surface area for absorption.

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