in the colon resistant starch is digested by
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ATI LPN

Nutrition ATI Proctored Exam

1. How is resistant starch digested in the colon?

Correct answer: A

Rationale: In the colon, resistant starch is digested by bacterial fermentation. The correct answer is A. During this process, short-chain fatty acids are produced. Pancreatic amylase, as mentioned in choice B, is responsible for breaking down starch in the small intestine, not in the colon. Choice C, hydrochloric acid, functions in the stomach to aid in the digestion of proteins, not starch. Villi and microvilli, as stated in choice D, are structures in the small intestine that absorb nutrients; they do not participate in the digestion of resistant starch in the colon.

2. Consuming 30 g of which nutrient would provide the highest number of kcalories?

Correct answer: B

Rationale: The correct answer is B, 'Fat.' Fat provides 9 kcalories per gram, which is more than carbohydrates and proteins that provide 4 kcalories per gram each. Therefore, consuming 30 g of fat would yield a higher number of kcalories compared to the same amount of carbohydrates or proteins. Micronutrients do not significantly contribute to kcalories, making them an incorrect choice in this scenario.

3. Which type of carbohydrate helps maintain normal blood glucose levels during fasting periods?

Correct answer: D

Rationale: The correct answer is glycogen. Glycogen is a type of carbohydrate stored in the body that is broken down into glucose to help maintain blood glucose levels during fasting periods. Glucose (choice A) is the simplest form of sugar and is a product of carbohydrate digestion, not the stored form like glycogen. Galactose (choice B) is a monosaccharide found in dairy products. Dextrins (choice C) are intermediate products of starch digestion and not directly involved in maintaining blood glucose levels during fasting.

4. In which health problem is protein catabolism increased?

Correct answer: A

Rationale: Protein catabolism increases in conditions like severe burns because the body breaks down muscle protein to provide energy for healing. Severe obesity does not necessarily increase protein catabolism; instead, it is more related to excessive fat accumulation. Hypothyroidism may lead to a decrease in metabolic rate but does not directly increase protein catabolism. Food allergies trigger an immune response to specific foods but do not directly impact protein catabolism.

5. What triggers the release of cholecystokinin (CCK)?

Correct answer: B

Rationale: The correct answer is B: Presence of fat in the duodenum. Cholecystokinin (CCK) is released in response to the presence of fat in the duodenum to aid in digestion by stimulating the release of bile from the gallbladder and enzymes from the pancreas. Options A, C, and D are incorrect because CCK is specifically released in response to the presence of fat in the duodenum, not food in the stomach, acid chyme in the ileum, or bile in the gallbladder.

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