identify the bone with the greatest length in the human body
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ATI TEAS 7

TEAS Test 7 science

1. Which bone is the longest in the human body?

Correct answer: A

Rationale: The correct answer is the femur (thigh bone) as it is the longest bone in the human body, extending from the hip to the knee. It plays a vital role in supporting the body's weight and enabling movement. The humerus is located in the upper arm and is not as long as the femur. The tibia is the shin bone and is shorter than the femur. The scapula is the shoulder blade and is not the longest bone in the body, making it an incorrect choice.

2. What effect does doubling the net force applied to an object have on its acceleration, assuming mass remains constant?

Correct answer: A

Rationale: According to Newton's second law of motion, acceleration is directly proportional to the net force applied to an object when mass is constant. Therefore, if the net force is doubled, the acceleration of the object will also double. This relationship is expressed by the formula F=ma, where F is the net force, m is the mass, and a is the acceleration. When mass is constant, doubling the force applied will result in a proportional doubling of acceleration. Choices B, C, and D are incorrect because doubling the net force does not halve, maintain, or quadruple the acceleration; it directly and proportionally increases the acceleration.

3. What are substances that stimulate adaptive immunity called?

Correct answer: A

Rationale: Substances that stimulate adaptive immunity are called peptides. Peptides are short chains of amino acids that play a crucial role in activating the immune response by interacting with immune cells such as T cells and B cells. Peptides are important in antigen presentation and recognition, leading to the development of specific immune responses against pathogens. Choice B, phagocytes, are cells that engulf and digest pathogens but are not substances that stimulate adaptive immunity. Choice C, prions, are infectious agents made of misfolded proteins and are not related to stimulating adaptive immunity. Choice D, platelets, are cell fragments involved in blood clotting and have no direct role in stimulating adaptive immunity.

4. What is the term for a genetic disorder caused by a mutation in a mitochondrial gene?

Correct answer: D

Rationale: A) Autosomal dominant disorder: This type of genetic disorder is caused by a mutation in one copy of an autosomal gene. It is not related to mitochondrial gene mutations. B) Autosomal recessive disorder: This type of genetic disorder is caused by mutations in both copies of an autosomal gene. It is not related to mitochondrial gene mutations. C) Sex-linked disorder: This type of genetic disorder is caused by mutations in genes located on the sex chromosomes (X or Y). It is not related to mitochondrial gene mutations. D) Mitochondrial disorder: Mitochondrial disorders are genetic disorders caused by mutations in genes located in the mitochondria, the energy-producing structures within cells. These disorders are inherited maternally and can affect various organs and systems in the body due to the role of mitochondria in energy production.

5. In an SN2 reaction, what affects the rate of the reaction?

Correct answer: D

Rationale: In an SN2 reaction, the rate of the reaction is affected by both the concentration of the nucleophile and the electrophile. The rate-determining step involves the nucleophile attacking the electrophile, so the concentrations of both species will impact the reaction rate. Increasing the concentration of the nucleophile increases the frequency of nucleophilic attacks, while increasing the concentration of the electrophile provides more opportunities for the nucleophile to react. Therefore, the correct answer is that both the concentration of the nucleophile and the electrophile affect the rate of the SN2 reaction. Choices A, B, and C are incorrect as they do not consider the interplay between the nucleophile and the electrophile in determining the overall reaction rate in an SN2 mechanism.

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