the following epithelium lines the inner surface of the oral cavity providing a barrier against external insults which type of epithelium best fits th
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ATI TEAS 7

ATI TEAS 7 science review

1. Which type of epithelium lines the inner surface of the oral cavity, providing a barrier against external insults?

Correct answer: B

Rationale: The oral cavity is constantly exposed to mechanical stress and potential injury from food particles and other external factors. To protect against these insults, a strong and resilient epithelium is needed. Stratified squamous epithelium is the best choice for lining the oral cavity due to its multiple layers of cells. This type of epithelium provides a robust barrier that can withstand abrasion and protect the underlying tissues. Simple squamous epithelium (choice A) is too thin and not suitable for a protective function. Simple columnar epithelium (choice C) is more suited for absorption and secretion functions, not protection against mechanical stress. Pseudostratified columnar epithelium (choice D) is found in respiratory passages and functions in mucociliary clearance, not in providing a protective barrier against external insults in the oral cavity.

2. What are the four main regions of the stomach?

Correct answer: B

Rationale: The correct answer is B: Cardia, fundus, body, pylorus. The four main regions of the stomach are the cardia (where food enters), fundus (upper part), body (largest part), and pylorus (exit part to the small intestine). These regions play specific roles in the digestion process. Choice A is incorrect because the cecum is part of the large intestine, not the stomach. Choice C lists parts of the small intestine, not the stomach. Choice D is incorrect as the ileum is the last part of the small intestine, not a region of the stomach.

3. Following successful heart transplant surgery, the patient's body will need to take medications to:

Correct answer: A

Rationale: After heart transplant surgery, the patient's body will need to take medications to suppress the immune system and prevent rejection of the new heart. This is essential to ensure that the body does not identify the transplanted heart as a foreign entity and mount an immune response against it, which could lead to transplant rejection and failure. Choices B, C, and D are incorrect because post-heart transplant medications primarily focus on preventing rejection by suppressing the immune system, rather than reducing blood pressure, dissolving blood clots, or stimulating the growth of new heart tissue.

4. What is the role of RNA interference in silencing genes?

Correct answer: C

Rationale: RNA interference (RNAi) is a biological process that involves the silencing of gene expression by inhibiting the translation of mRNA or by degrading mRNA molecules. This mechanism plays a crucial role in regulating gene expression and can be used to selectively silence specific genes. By interfering with the expression of specific genes, RNAi can downregulate protein production from those genes. Therefore, option C, 'Inhibits the expression of specific genes,' is the correct role of RNA interference in silencing genes. Options A, B, and D are incorrect because RNA interference does not increase protein production, promote gene mutation, or repair damaged DNA; its primary function is to inhibit gene expression by degrading or interfering with mRNA.

5. Diabetes mellitus is a chronic condition affecting blood sugar control. What is the main type of hormone deficiency associated with type 1 diabetes?

Correct answer: A

Rationale: The correct answer is A: Insulin. Type 1 diabetes is characterized by the body's inability to produce insulin, the hormone responsible for regulating blood sugar levels. Insulin deficiency in type 1 diabetes leads to high blood sugar levels as the body cannot effectively utilize glucose for energy. Glucagon (Choice B) is a hormone that works opposite to insulin, raising blood sugar levels. Estrogen (Choice C) and testosterone (Choice D) are not directly involved in blood sugar control or the pathophysiology of type 1 diabetes.

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