ATI TEAS 7
TEAS 7 science quizlet
1. How does the structure of centromeres contribute to chromosome movement during mitosis?
- A. The centromere provides a binding site for spindle fibers, allowing chromosomes to be attached and manipulated.
- B. The centromere serves as a dividing point between sister chromatids, ensuring their separation during anaphase.
- C. The centromere maintains chromosome stability by preventing chromosomal breaks and rearrangements.
- D. The centromere plays a role in DNA replication, ensuring accurate copying of the genetic material.
Correct answer: A
Rationale: A) The centromere provides a binding site for spindle fibers, which are microtubules that help move chromosomes during cell division. This attachment allows the chromosomes to be pulled towards opposite poles of the cell during mitosis. Therefore, the structure of centromeres directly contributes to chromosome movement during mitosis by facilitating the attachment and manipulation of chromosomes by the spindle fibers. B) While the centromere does serve as a dividing point between sister chromatids, ensuring their separation during anaphase, this function is more related to the segregation of chromosomes rather than their movement. C) The centromere does play a role in maintaining chromosome stability by ensuring proper chromosome segregation, but it is not primarily responsible for preventing chromosomal breaks and rearrangements. D) The centromere is not directly involved in DNA replication. Its main function is
2. Which gland located in the neck is responsible for regulating metabolism, growth, and development?
- A. Parathyroid gland
- B. Thyroid gland
- C. Pituitary gland
- D. Thymus gland
Correct answer: B
Rationale: The thyroid gland, located in the neck, is responsible for regulating metabolism, growth, and development. It produces hormones such as thyroxine (T4) and triiodothyronine (T3) that play a crucial role in these functions. The parathyroid gland regulates calcium levels, the pituitary gland controls various hormonal functions, and the thymus gland is involved in immune system development. Therefore, choices A, C, and D are incorrect as they have different functions compared to the thyroid gland.
3. Which of the following is an example of a long bone in the human body?
- A. Carpals
- B. Femur
- C. Tarsals
- D. Metacarpals
Correct answer: B
Rationale: The correct answer is B, Femur. The femur is the longest and strongest bone in the human body, located in the thigh region. Long bones are characterized by their elongated shape, such as the femur, which provides support, strength, and mobility. Choices A, C, and D are incorrect because carpal bones (A) are short bones found in the wrist, tarsal bones (C) are short bones found in the ankle, and metacarpal bones (D) are classified as long bones, but they are shorter than the femur and mainly found in the hand.
4. What is the term for the division of the cytoplasm?
- A. Cytokinesis
- B. Mitosis
- C. Meiosis
- D. Apoptosis
Correct answer: A
Rationale: Cytokinesis is the correct term for the division of the cytoplasm during cell division. It is the final stage of cell division, following either mitosis or meiosis, where the cytoplasm of a parent cell is divided into two daughter cells. Mitosis is the process of nuclear division, while meiosis is a type of cell division that results in four daughter cells with half the number of chromosomes of the parent cell. Apoptosis, on the other hand, is programmed cell death and not the division of the cytoplasm.
5. Which protein complex in the sarcomere is responsible for the thick filament?
- A. Actin
- B. Myosin
- C. Tropomyosin
- D. Troponin
Correct answer: B
Rationale: Myosin is the protein complex responsible for the thick filament in the sarcomere. It interacts with actin, another protein in the sarcomere, to generate the sliding movement that results in muscle contraction. Actin is a thin filament protein that interacts with myosin to create the sliding filament mechanism. Tropomyosin and troponin are regulatory proteins that regulate muscle contraction by controlling the interaction between actin and myosin, rather than directly being responsible for the thick filament.
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